Cell Line-Derived Xenograft (CDX) Models

Fast and Scalable Cell Line-Derived Xenograft (CDX) Models

LIDE’s Cell Line-Derived Xenograft (CDX) models offer a high-throughput, cost-effective in vivo platform for evaluating oncology drug candidates. Derived from well-characterized cancer cell lines, these models are ideal for rapid efficacy testing, biomarker investigation, and early pharmacology studies.

Why choose CDX models?

CDX models are established by transplanting human cancer cell lines into immunodeficient mice, offering several key advantages:

  • Speed & Efficiency: Faster engraftment and tumor development compared to PDX — perfect for agile screening.
  • Highly Reproducible: Standardized tumor growth from immortalized lines ensures consistency across studies.
  • Cost-Effective: Lower resource requirements make CDX models ideal for initial dose and regimen studies.
  • Predictive Biology: They mirror many molecular and pharmacological traits of the source tumors, including treatment responses

LIDE’s Expansive CDX Portfolio

We curate one of the most diverse and rapidly growing CDX collections-ideal for early-stage R&D. 120+ established models spanning major tumor types:

Cancer TypeCell Lines# of Available CDX Models
Bladder Cancer5637, RT112, SW780, T244
Breast CancerBT474, HCC1806, HCC-1954, JIMT-1, MCF-7, MDA-MB-231, MDA-MB-453, MDA-MB-468, MFM-223, SUM149PT, ZR-75-111
Cervical CancerC-33A, HeLa, SK-UT-13
Colon CancerCOLO 205, DLD-1, GP2D, HCT-116, HT-29, LS513, LOVO, RKO, SW1463, SW48, SW480, SW620, SW83713
Esophageal CancerKYSE-301
FibrosarcomaHT10801
Gastric CancerAGS, KATO III, MKN1, MKN45, NCI-N87, NCI-N87-Claudin 18.2, NUGC-3, SNU-16, SNU-59
Liver CancerHep G2, Hep3B, Huh7, MHCC97H, SNU-3985
Lung CancerA549, Clau-1, Clau-3, DMS114, EBC-1, HCC827, MSTO-211H, NCI-H1299, NCI-H1373, NCI-H1975, NCI-H2009, NCI-H209, NCI-H2122, NCI-H226, NCI-H292, NCI-H358, NCI-H441, NCI-H460, NCI-H524, NCI-H596, NCI-H838, PC-9, SHP-7723
Lymphoma & LeukemiaDaudi, Jurkat clone E6-1, Karpars-299, K-562, KG-1, MOLT-4, MOLM-13, MOLM-16, MV-4-11, NALM-6, OCI-AML2, OCI-LY3, Ramos(RA 1), Raji, RPMI 8226, RS4;11, Toledo17
MelanomaA-375, A-4312
Pancreas CancerASPC-1, BXPC-3, Capan-2, HPAC, MIA PaCa-2, PANC-1, PK-597
Prostate Cancer22Rv1, DU145, LNCaP Clone FGC, PC-34
Renal Carcinoma786-O, A-498, SK-NEP-13
Ovarian CancerSK-OV-3, PA-1, A2780, ES-2, OVCAR-35
GlioblastomaU87-MG, U251, LN-2293
Multiple MyelomaMOLP-81
OsteosarcomaSAOS-21
RetinoblastomaWERI-Rb-1, Y-792
Head and Neck CancerSCC090, CAL 27, CAL-33, KYES450, FaDu5
RhabdomyosarcomaRD-ES1

LIDE’s CDX models are compatible with humanized mouse systems, enabling immunotherapy evaluation within human-like immune contexts.

Additionally, LIDE has 220+ human cancer cell lines available in house for CDX development, enriched with drug-resistant variants and clinically relevant phenotypes.

ApplicationWhy CDX Makes a Difference
High-Throughput ScreeningEfficiently compare multiple drug candidates across fast-growing models
Dose & Schedule OptimizationExplore dose-response dynamics before committing to resource-intensive PDX
Standard-of-Care BenchmarkingTest new compounds directly against established therapies
Biomarker DiscoveryLink molecular features to in vivo drug response across uniform models
Proof of Concept & MoA ValidationFast, reliable efficacy readouts to inform next-phase decisions
Immune-Oncology Testing (Humanized)Model checkpoint inhibitors, CAR-Ts, and ADCs with functional immune systems

Differentiating CDX vs PDX: Why Both Matter

While PDX models preserve tumor heterogeneity and deliver high translational fidelity, they require months to establish and scale. CDX models, on the other hand, offer:

  • Rapid experimentation cycles
  • Lower compound and animal usage
  • Immediate, reproducible results
  • Ideal for early phase selection before deploying PDX or orthotopic validation

Used sequentially, LIDE’s CDX + MiniPDX® + PDX platform form a powerful, tiered preclinical strategy that balances speed, accuracy, and scale.